Additional file 4: Table S5. of Systematic analysis of chromatin interactions at disease associated loci links novel candidate genes to inflammatory bowel disease
*Derived from non-linear fitting of data recorded to ΔFemission = Fmax×[L]/(Kd+[L]) (single site saturation binding curve). a, no change in Femission over background in response to ligand additions.
MS is a primary demyelinating disease of the central nervous system. The cause is unknown and pathological hallmarks include plaque formation with immune cell infiltration. A microarray analysis of ch
RAD21 ChIA-PET in human ARPE-19 cells For data usage terms and conditions, please refer to http://www.genome.gov/27528022 and http://www.genome.gov/Pages/Research/ENCODE/ENCODE_Data_Use_Policy_for_Ex
Expanded CAG/CTG repeats underlie thirteen neurological disorders, including myotonic dystrophy type 1 (DM1) and Huntington's disease (HD). Upon expansion, CAG/CTG repeat loci acquire heterochromatic
