The transcription program of Runx3 in natural killer cells and CD8+ T cells

ChEBI2013-11-18 更新2026-07-21 收录

细胞转录调控

Runx3功能研究

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-50131 数据链接链接失效反馈

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The transcription program of Runx3 in natural killer cells and CD8+ T cells

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2013-11-18

The Hippo Pathway Prevents YAP/TAZ–Driven Hypertranscription and Controls Neural Progenitor Number [ChIP-seq]. The Hippo Pathway Prevents YAP/TAZ–Driven Hypertranscription and Controls Neural Progenitor Number [ChIP-seq]

细胞转录调控

Hippo信号通路

The Hippo pathway controls the activity of YAP/TAZ transcriptional coactivators through a kinase cascade. Despite the critical role of this pathway in tissue growth and tumorigenesis, it is not fully

NIAID Data Ecosystem60

Transcriptional Reversion of Cardiac Myocyte Fate During Mammalian Cardiac Regeneration.

心脏再生机制

细胞转录调控

Transcriptional Reversion of Cardiac Myocyte Fate During Mammalian Cardiac Regeneration.

ChEBI2014-12-20 更新20

Concerted repression of an immunoglobulin heavy-chain enhancer, 3' alpha E(hs1,2).

细胞转录调控

免疫球蛋白增强子功能

The transcription factor, B-cell-specific activator protein (BSAP), represses the murine immunoglobulin heavy-chain 3' enhancer 3' alpha E(hs1,2) in B cells. Analysis of various 3'alpha E deletional c

PubMed Central1996-04-30 更新10

Gene expression data from mouse conventional dendritic cells (cDC) [Batf, Batf3 DKO]

细胞转录调控

BATF转录因子功能

We used microarrays to understand roles of BATF proteins in the regulation of cDC-specific gene expression. Splenic cDCs were sort-purified from WT or Batf–/– Batf3–/– mice for RNA extraction and hybr

NIAID Data Ecosystem50

RNA-seq analysis of WT and NCOR1f/f,Cd4Cre in vitro-generated Th17 cells

细胞转录调控

NCOR1辅因子功能

The differentiation of naïve CD4+ T cells into T helper (Th) subsets is key for a functional immune response and has to be tightly controlled by transcriptional and epigenetic processes. However, the

NIAID Data Ecosystem20

The transcription program of Runx3 in natural killer cells and CD8+ T cells

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