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A Splice Variant of HER2 Activates Key Signaling Cascades and Evokes Mammary Tumors and Metastases
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2025-01-24
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Explosure of breast cancer cells promotes estrogen-dependent and -independent cell proliferation mechanisms tied to clinical outcomes identified in ER+ breast cancers
Differentially expressed genes identified in MCF-7 cells treated to 0.1% ethanol concentration. Total RNA was generated from MCF-7 breast cancer cells starved of estrogen in 5% charcoal filtered FBS.
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Data_Sheet_1_Fra-1 and c-Fos N-Terminal Deletion Mutants Impair Breast Tumor Cell Proliferation by Blocking Lipid Synthesis Activation.pdf
Tumor cells require high rates of lipid synthesis to support membrane biogenesis for their exacerbated growth. The only two proteins known that activate phospholipid synthesis are Fra-1 and c-Fos, two
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Co-regulated gene expression by estrogen receptor-a and liver receptor homolog-1 is a feature of the estrogen response in breast cancer cells
Co-regulated gene expression by estrogen receptor-a and liver receptor homolog-1 is a feature of the estrogen response in breast cancer cells
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Global studies of Zeb1 on breast cancer.
we knockout zeb1 in the mouse mammary epithelial cells to study the role of zeb1 in tumor initiation, progression and the pathological mechanisms.
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Non-enzymatic SETD1A activity drives breast cancer cell proliferation via cyclin K. (ChIP-seq)
Breast cancer is the most common cancer in women worldwide. SETD1A, a histone H3 lysine-4 methyltransferase, is associated with poor prognosis in breast cancer. While both its enzymatic and non-enzyma
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