Nineteen transcript isoforms of eight disrupted cadherin alleles in seven pink bollworm larvae from two populations in India: Anand, Gujarat (AGJ) and Khandwa, Madhya Pradesh (KMP).

aMutations shown in bold cause premature stop codons.bRegion of cadherin protein where major mutations occur (see Figure 2).cThe 478-bp deletion found in r5A, r5B and r5C is caused by insertion of 3,120 bp similar to transposons (Table 2), causing the loss of exons 21–24 from gDNA and cDNA.dThe 3-bp deletion in r5B and r12B is caused by mis-splicing, occurs at exon-intron splice junction 1, and is found in both r and s PgCad1 alleles.egDNA from AGJ-2 was not available to compare with cDNA, but the absence of exons 8–13 occurs exactly at the exon-intron junctions, suggesting that mis-splicing occurred.fr9A includes A-to-G (I) RNA editing at base position 2,289 and results in the introduction of a premature stop codon (see Fig. 4). The 165-bp deletion causes the loss of exon 32.gThe 23-bp deletion corresponds to the final 23 nucleotides of exon 20 in cDNA clone KMP-6_3.hThe single base insertion introduces a premature stop codon and truncates the mRNA transcript in CR11.iThe 118-bp deletion causes the loss of exon 11 resulting in the introduction of a premature stop codon and truncates the mRNA transcript in CR4.jThe 11-bp deletion occurs in the membrane signal sequence of r12C and r12D transcripts resulting in the introduction of a premature stop codon.kThe 148-bp deletion causes the loss of exon 5 in mRNA transcript between the membrane signal sequence and CR1.lThe 230-bp deletion causes the loss of exons 11–12 in mRNA transcript found in CR4.