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The dysregulated inclusion of BOLA3 exon 3 promoted by HNRNPC accelerates the progression of esophageal squamous cell carcinoma in esophageal squamous cell carcinoma

NIAID Data Ecosystem2026-05-01 收录

食管鳞状细胞癌分子病理学

RNA剪接调控

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE235538 数据链接链接失效反馈

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This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

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创建时间：

2024-01-06

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Although mutations in DNA are the best-studied source of neoantigens that determine response to immune checkpoint blockade, alterations in RNA splicing within cancer cells could similarly result in ne

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iCLIP-based modeling uncovers 3' splice site definition: how U2AF65 specificity relies on regulation by co-factors

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Alternative splicing generates distinct mRNA isoforms and is crucial for proteome diversity in eukaryotes. The RNA-binding protein (RBP) U2AF65 is central to splicing decisions, as it recognizes 3' sp

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Nuclear RNA-seq of siRNA-mediated METTL3 knockdown. Nuclear RNA-seq of siRNA-mediated METTL3 knockdown

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To identify changes in splicing patterns following METTL3 knockdown, RNA-sequencing was performed on nuclear RNA extracted from cells transfected with METTL3-targeting siRNA or a non-targeting siRNA (

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The impact of Prp18p on splicing fidelity and efficiency in budding yeast

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Fidelity of 3´-splice site (3´SS) selection by the spliceosome is critical for proper gene expression but is a daunting task considering the low complexity of the 3´SS consensus YAG. Here we show that

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The dysregulated inclusion of BOLA3 exon 3 promoted by HNRNPC accelerates the progression of esophageal squamous cell carcinoma in esophageal squamous cell carcinoma

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