GDF15-GFRAL orchestrates glutathione synthesis via NRF2-elicited GCLC activation to impedes cuproptosis
收藏NIAID Data Ecosystem2026-05-01 收录
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https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE225628
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This study identified a non-canonical function of GDF15-GFRAL pathway that plays oncogenic roles and modulates cuproptosis by mediating GSH addiction in SCC. To identify the critical regulatory of cuproptosis, we performed RNA-seq in parental and DTP (EC) cells. RNA-seq of parental and drug tolerant persister (DTP) KYSE450 cells
创建时间：
2023-08-01
