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DNA methylation landscape and signature of CD4+ lymphocytes of lamina propria in Crohn's patients by reduced representation and bisulfite sequencing

NIAID Data Ecosystem2026-05-01 收录

肠道免疫表观遗传学

克罗恩病甲基化图谱

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE184314 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

应用场景：

创建时间：

2023-12-01

组蛋白修饰

肠道免疫表观遗传学

For data usage terms and conditions, please refer to http://www.genome.gov/27528022 and http://www.genome.gov/Pages/Research/ENCODE/ENCODE_Data_Use_Policy_for_External_Users_03-07-14.pdf https://www.e

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Histone ChIP-seq from Peyer's patch (ENCSR912XAL)

组蛋白修饰

肠道免疫表观遗传学

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Impact of the gut microbiota on enhancer accessibility in gut intraepithelial lymphocytes

肠道免疫表观遗传学

微生物-宿主互作

The gut microbiota impacts many aspects of host biology including immune function. One hypothesis is that microbial communities induce epigenetic changes with accompanying alterations in chromatin acc

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ChIP-seq analysis of BATF in ILC3s sorted from small intestine of Rag1-/- mice [CUT&TAG ChIPseq]. ChIP-seq analysis of BATF in ILC3s sorted from small intestine of Rag1-/- mice [CUT&TAG ChIPseq]

肠道免疫表观遗传学

ILC3细胞调控机制

Group 3 innate lymphoid cells (ILC3s) are crucial for the maintenance of host-microbiota homeostasis in gastrointestinal mucosal tissues. The mechanisms that maintain lineage identity of intestinal IL

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Alterations to chromatin in intestinal macrophages link IL-10 deficiency to inappropriate inflammatory responses

肠道免疫表观遗传学

结肠炎机制

Intestinal macrophages (IMs) are uniquely programmed to tolerate exposure to bacteria without mounting potent inflammatory responses. The cytokine IL-10 maintains the macrophage anti-inflammatory resp

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DNA methylation landscape and signature of CD4+ lymphocytes of lamina propria in Crohn's patients by reduced representation and bisulfite sequencing

资源简介：

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