Effect of retinoic acid on gene expression in CD4 cells stimulated with anti CD3 and CD28

ChEBI2016-02-29 更新2026-07-12 收录

细胞免疫调控

药物干预分子机制

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Effect of retinoic acid on gene expression in CD4 cells stimulated with anti CD3 and CD28

创建时间：

2016-02-29

THP-1 cell study: RA and TNF Treatments; RA time course. Homo sapiens

细胞免疫调控

视黄酸时间序列分析

The study consisted of two experiments. The hypothesis tested was that RA and tumor necrosis factor (TNF)-alpha would independently and synergistically regulate the expression of genes in THP-1 human

NIAID Data Ecosystem60

Table_2_Human Lymphoid Stromal Cells Contribute to Polarization of Follicular T Cells Into IL-4 Secreting Cells.doc

细胞免疫调控

滤泡淋巴瘤微环境

Fibroblastic reticular cells (FRCs) are the specialized lymphoid stromal cells initially identified as triggering T-cell recruitment and dynamic motion in secondary lymphoid organs. Interestingly, FRC

NIAID Data Ecosystem50

Regulatory RNA inhibition in human phagocytes using cell-penetrating PNA oligonucleotides. Regulatory RNA inhibition in human phagocytes using cell-penetrating PNA oligonucleotides

RNA靶向治疗

细胞免疫调控

Here, we determined the ability of peptide nucleic acid (PNA) oligomers, coupled to different cell-penetrating peptides (CPPs), to interfere in regulatory RNA circuits of human blood-derived leukocyte

NIAID Data Ecosystem30

Neutrophils stimulation with AVP promote IL36G release

细胞免疫调控

造血过程

We found that neutrophils were homing back to bone marrow and induced myelopoiesis after AVP treatment.This study investigated how neutrophils promoted HSC differentiation by comparing the expression

NIAID Data Ecosystem60

Deciphering the importance of the palindromic architecture of the immunoglobulin heavy chain 3’ regulatory region.. Mus musculus

细胞免疫调控

免疫球蛋白重组

The IgH 3’ regulatory region (3’RR) controls class switch recombination (CSR) and somatic hypermutation (SHM) in B cells. The mouse 3’RR contains four enhancer elements with hs1,2 flanked by inverted

NIAID Data Ecosystem40

Effect of retinoic acid on gene expression in CD4 cells stimulated with anti CD3 and CD28

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