Rev-Erbs repress macrophage gene expression by inhibiting enhancer-directed transcription

ChEBI2013-06-05 更新2026-07-21 收录

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巨噬细胞免疫

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-45914 数据链接链接失效反馈

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Rev-Erbs repress macrophage gene expression by inhibiting enhancer-directed transcription

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2013-06-05

ToxR bacterial transcriptional regulator bound to 25 bp toxT promoter DNA

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DNA相互作用

ToxR bacterial transcriptional regulator bound to 25 bp toxT promoter DNA Descriptor: Cholera toxin transcriptional activator, DNA (25-MER) Authors: Canals, A, Pieretti, S, Muriel, M, El Yaman, N, Fab

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Nucleoporin 153 links nuclear pore complex to chromatin architecture by mediating CTCF and cohesin binding [RNA-Seq]

染色质三维构象调控

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Establishment of cell lineage specification, maintenance of cellular states and cellular responses to developmental cues rely on gene regulation and spatial genome organization during early developmen

NIAID Data Ecosystem50

A cohesin traffic pattern genetically linked to gene regulation [ChIP-Seq]

cohesin介导的染色质环挤压

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Cohesin-mediated loop extrusion folds interphase chromosomes at the ten to hundreds kilobases scale. This process produces structural features such as loops and topologically associating domains. We i

NIAID Data Ecosystem10

Table S1 from Disruption of TFIIH activities generates a stress gene expression response and reveals possible new targets against cancer

癌症靶向治疗

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Disruption of the enzymatic activities of the transcription factor TFIIH by the small molecules Triptolide (TPL) or THZ1 could be used against cancer. Here, we used the MCF10A-ErSrc oncogenesis model

Figshare2020-06-01 更新50

Dual regulation of open-complex formation and promoter clearance by Arc explains a novel repressor to activator switch.

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基因表达

In studies of variants of the P(ant) promoter of bacteriophage P22, the Arc protein was found not only to slow the rate at which RNA polymerase forms open complexes but also to accelerate the rate at

PubMed Central1996-08-20 更新30

Rev-Erbs repress macrophage gene expression by inhibiting enhancer-directed transcription

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