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Mithramycin Depletes Sp1 and Activates p53 to Mediate Senescence and Apoptosis of Pleural Mesothelioma Cells

ChEBI2015-01-07 更新2026-07-21 收录

肿瘤药物机制

细胞凋亡通路

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-64738 数据链接链接失效反馈

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Mithramycin Depletes Sp1 and Activates p53 to Mediate Senescence and Apoptosis of Pleural Mesothelioma Cells

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2015-01-07

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细胞凋亡通路

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细胞凋亡通路

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Propionibacterium acnes induces intervertebral disc degeneration by promoting nucleus pulposus cell apoptosis via the TLR2/JNK/mitochondrial-mediated pathway

椎间盘退变机制

细胞凋亡通路

Evidence suggests that intervertebral disc degeneration (IVDD) can be induced by Propionibacterium acnes (P. acnes), although the underlying mechanisms are unclear. In this study, we analyzed the path

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Apoptosis pathway analysis of genes from macropahges infected with Histoplasma capsulatum. Mus musculus

细胞凋亡通路

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Analyzed 84 genes from macrophages infected with Histoplasma capsulatum for changes in expression over 24 hours Overall design: Macrophages infected with Histoplasma capsulatum were analyzed for alter

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antitumor effect of berberine from perspective of gene expression

肿瘤药物机制

基因表达分析

to explore the pro-carcinogenic property of high-fat diet in AOM/DSS induced CRC mice model and to fathom how berberine inhibits the formation of adenocarcinoma induced by high-fat diet. We then perfo

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Mithramycin Depletes Sp1 and Activates p53 to Mediate Senescence and Apoptosis of Pleural Mesothelioma Cells

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