RNA-seq of L3.6pl cells treated with CI994, TW9, and CI994 + JQ1

NIAID Data Ecosystem2026-05-01 收录

转录组药物响应

癌细胞基因表达分析

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https://www.ncbi.nlm.nih.gov/sra/ERP110942 数据链接链接失效反馈

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资源简介：

Evaluation of gene expression profiles upon treatment with CI994, TW9, and CI994 + JQ1 for 24 hours treatment

创建时间：

2023-10-13

Transcriptional profiling of M. tuberculosis treated with DG167 and INH

结核分枝杆菌药物机制

转录组药物响应

We report that combined treatment with both DG167 and INH activates a cellular response associated with loss of persistence and induction of cidality that is distinct from the cellular response induce

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Effect of ribociclib on breast cancer cells

乳腺癌药物机制

转录组药物响应

To study mechanisms underlying response to ribociclib. Overall design: Examination of breast cancer cell transcriptome following treatment with ribociclib (600 nM).

NIAID Data Ecosystem50

Supplementary Table S4 from C/EBPα Confers Dependence to Fatty Acid Anabolic Pathways and Vulnerability to Lipid Oxidative Stress–Induced Ferroptosis in FLT3 -Mutant Leukemia

FLT3-ITD突变白血病

转录组药物响应

Table S4: GSEA results in FLT3-ITD PDX AML cells ex vivo-treated with QUIZ or Veh.

NIAID Data Ecosystem40

Transcriptome analysis of CPUL1 on regulating hepatocellular carcinoma growth via autophagy-related cell death

肝细胞癌药物

转录组药物响应

Compound CPUL1 is one of the phenazine analogues. Phenazines is one nitrogen-containing hetercyclic compound that is natural or synthetized, possessing many pharmacological benefits, including antimal

NIAID Data Ecosystem50

Suppression of adaptive responses to targeted cancer therapy by transcriptional repression [RNA-seq]. Homo sapiens

肿瘤耐药机制

转录组药物响应

Large-scale genomic profiling efforts have facilitated the characterization of molecular alterations in cancers and aided the development of targeted kinase inhibitors for a wide array of cancer types

NIAID Data Ecosystem50

RNA-seq of L3.6pl cells treated with CI994, TW9, and CI994 + JQ1

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