To investigate the functions of HPV16 E5, we established empty vector or HPV16 E5-transduced CAL-27 and DC2.4 cell lines. We then performed gene expression profiling analysis using data obtained from

NIAID Data Ecosystem30

Epidermal cancer associated with expression of human papillomavirus type 16 E6 and E7 oncogenes in the skin of transgenic mice.

HPV致癌机制

转基因小鼠肿瘤模型

Certain "high-risk" anogenital human papillomaviruses (HPVs) have been associated with the majority of human cervical carcinomas. In these cancers, two papillomaviral genes, E6 and E7, are commonly ex

PubMed Central1993-06-15 更新20

Long non-coding RNA FAM83H-AS1 is regulated by human papillomavirus 16 E6 independently of p53 in cervical cancer cells

HPV致癌机制

lncRNA

Global long non-coding RNA (lncRNA) expression profile in primary human keratinocytes post-expression of high-risk HPV-16 E6 oncoprotein Purpose: The goal of this study is to utlize high-throughput RN

NIAID Data Ecosystem30

Identification of deregulated pathways, key regulators, and novel miRNA-mRNA interactions in HPV-mediated transformation. [CGH cell lines-Agilent]. Identification of deregulated pathways, key regulators, and novel miRNA-mRNA interactions in HPV-mediated transformation. [CGH cell lines-Agilent]

HPV致癌机制

分子生物学

Next to a persistent infection with high-risk human papillomavirus (HPV), molecular changes are required for the development of cervical cancer. To identify which molecular alterations drive carcinoge

NIAID Data Ecosystem20

HPV-16 and -18 cervical cancer Transcriptome. Homo sapiens

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