CKD original data.sav

Our research found as follows: Even in acute coronary syndrome patients treated by similar lipid-lowering statin therapy, the normalized total atheroma volume of non-culprit subclinical coronary atherosclerosis increased significantly, with the calcium component increasing in mild chronic kidney disease (CKD) compared with that in non-CKD at the 1-year follow-up. The morphological changes in calcium lesions showed greater maximal calcification thickness and more lumen protrusion in mild CKD compared to non-CKD. The major adverse cardiac events (MACEs) related to non-culprit subclinical coronary atherosclerosis were significantly higher in mild CKD than in non-CKD at the 5-year follow-up even with the same lipid-lowering therapy at 1 year.