KrasG12D partially compensates for the loss of beta-catenin in MLL-AF9 AML. Mus musculus
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https://www.ncbi.nlm.nih.gov/bioproject/PRJNA213401
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资源简介：
The Wnt/beta-catenin pathway is required for the development of leukemia stem cells in MLL-AF9 AML. We evaluated the dependance on beta-catenin for KrasG12DMLL-AF9 leukemia. Overall design: Lin-Kit+ bone marrow cells obtained from mice transplanted with primary MLL-AF9 leukemia cells and KRasG12DMLL-AF9 leukemia cells were assessed for gene expression in the presence or absence of beta-catenin
创建时间：
2013-07-26
