Peptide OM-LV20 Demonstrates Neuroprotective Effects by Attenuating Mitochondrial-Mediated Neuronal Apoptosis in Traumatic Brain Injury (TBI) Mice
收藏科学数据银行2025-10-03 更新2026-04-23 收录
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https://www.scidb.cn/detail?dataSetId=c1efdf115707496a87842527bdc6765a
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资源简介：
Traumatic Brain Injury (TBI) is a major global health concern, especially in people under 45, causing long-term motor and cognitive impairments with limited effective therapies. Peptide OM-LV20, from odorous frogs in Southwest China, shows neuroprotection in oxidative stress, spinal cord injury, and cerebral ischemia/reperfusion. This dataset uses RNA-seq to capture transcriptomic changes in TBI mice’s frontal cortex and hippocampus after OM-LV20 intervention. It supports studying OM-LV20’s neuroprotective mechanism via mitochondria-mediated neuronal apoptosis and serves as a reusable transcriptomic resource for TBI research and drug screening.
提供机构：
Kunming Medical University; Min-Nan Lu; Sai-Ge Yin; Yunnan Province Third People's Hospital; Chen-Jun Ye; The Third People's Hospital of Honghe Hani and Yi Autonomous Prefecture
创建时间：
2025-10-03
