Usv101 is a key regulator of cryptococcal virulence and determinant of host response

ChEBI2016-03-31 更新2026-07-12 收录

隐球菌毒力机制

宿主-病原体相互作用

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-69532 数据链接链接失效反馈

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Usv101 is a key regulator of cryptococcal virulence and determinant of host response

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2016-03-31

Additional file 1: of Identification of new loci involved in the host susceptibility to Salmonella Typhimurium in collaborative cross mice

宿主-病原体相互作用

遗传学

Table S1. Individual organ bacterial loads at day 4 post-infection with Salmonella Typhimurium. Individual values for each animal tested are given: animal number (N), strain, alias (collected from UNC

DataCite Commons2024-03-25 更新90

LMP1 Protein binds to TRAF3 as a structural CD40

病毒蛋白结构与功能

宿主-病原体相互作用

LMP1 Protein binds to TRAF3 as a structural CD40 Descriptor: Latent membrane protein 1, TNF receptor associated factor 3 Authors: Wu, S.D, Xie, P, Welsh, K, Li, C, Ni, C.-Z, Zhu, X, Reed, J.C, Sattert

Protein Data Bank Japan2024-02-14 更新40

The top 40 up- and down-regulated genes in hMDMs infected by either wild type or an isogenic ankB L. pneumophila mutant strain compared to uninfected hMDMs at 8 h post-infection.

宿主-病原体相互作用

基因表达谱分析

The top 40 up- and down-regulated genes in hMDMs infected by either wild type or an isogenic ankB L. pneumophila mutant strain compared to uninfected hMDMs at 8 h post-infection.

NIAID Data Ecosystem10

Coiling Phagocytosis of Trypanosomatids and Fungal Cells

细胞吞噬机制

宿主-病原体相互作用

Coiling phagocytosis has previously been studied only with the bacteria Legionella pneumophila and Borrelia burgdorferi, and the results were inconsistent. To learn more about this unconventional phag

PubMed Central50

Host chaperones can define the fitness of adaptive influenza variants

流感病毒

宿主-病原体相互作用

The threat of viral pandemics demands a comprehensive understanding of evolution at the host-pathogen interface. Here, we systematically show that the accessibility of adaptive mutations in influenza

NIAID Data Ecosystem30

Usv101 is a key regulator of cryptococcal virulence and determinant of host response

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