Microarray experiments on DCs challenged with fungi and single agonists.

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Microarray experiments on DCs challenged with fungi and single agonists.

创建时间：

2016-10-31

Gene expression data from Wild Type and Nlrp10 deficient dendritic cells treated with or without LPS. Mus musculus

树突状细胞免疫应答

基因表达谱分析

Nlrp10-deficient mice have a profound defect in helper T cell-driven immune responses. T cell priming is impaired due to a defect in the emigration of a dendritic cells from inflamed tissue and antige

NIAID Data Ecosystem90

Bacterial Infection Remodels the DNA Methylation Landscape of Human Dendritic Cells (wtRNA-Seq)

细菌感染表观遗传重塑

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DNA methylation is an epigenetic mark thought to be robust to environmental perturbations on a short time scale. Here, we challenge that view by demonstrating that the infection of human dendritic cel

NIAID Data Ecosystem70

PANTHER GO enrichment analyses.

基因功能富集分析

感染基因表达

Sheet S1 contains results of the statistical enrichment tests of the 222 significantly upregulated genes shared between N. parisii and N. ausubeli infected C. elegans. Sheet S2 contains results of the

Figshare2022-12-19 更新30

RNA-Seq analysis to profile the transcriptome of Bone Marrow Dendritic Cells exposed to different antigen delivery systems. Mus musculus

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疫苗学转录组分析

Here we report the analysis of the transcriptome of bone marrow-derived dendritic cells exposed to the different antigen delivery systems to dissect the molecular mechanisms that orchestrate the immun

NIAID Data Ecosystem80

Divergent principles specifying inflammatory versus antiviral transcriptional programs in dendritic cells [RNA-seq]. Divergent principles specifying inflammatory versus antiviral transcriptional programs in dendritic cells [RNA-seq]

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TLR2信号通路

Stimulus-specific gene expression programs are enabled by enhancers, on which stimulus-regulated transcription factors (SRTFs) can land in a cell type- and stimulus-dependent manner. In this study, we

NIAID Data Ecosystem50

Microarray experiments on DCs challenged with fungi and single agonists.

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