Genome-scale chromatin interaction data for huntingtin from mouse striatal tissue

Transcriptional changes are an early feature of Huntington's disease (HD). We profiled genome-wide interaction sites for the huntingtin protein (HTT) using ChIP-sequencing from mouse striatal tissue at 4 months of age. We include replicate samples from CAG-expanded murine Htt (heterozygous Q111/+) and wildtype littermate controls. Overall design: ChIP-seq for HTT (huntingtin) in wild type and Q111/+ CAG-expanded knock-in mouse striatum at 4 months of age.