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Metastasis and Survival of Breast Cancer Stem Cells Mediated by Cytoskeleton Remodeling and PI3K/mTOR Signaling transcription factors

ChEBI2012-04-13 更新2026-07-21 收录

乳腺癌干细胞

mTOR信号通路

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-26621 数据链接链接失效反馈

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资源简介：

Metastasis and Survival of Breast Cancer Stem Cells Mediated by Cytoskeleton Remodeling and PI3K/mTOR Signaling transcription factors

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创建时间：

2012-04-13

乳腺癌干细胞

EMT与转移抑制

Triple-negative breast cancer (TNBC) is more aggressive and has poorer prognosis compared to other subtypes of breast cancer. Epithelial-to-mesenchymal transition (EMT) is a process in which epithelia

NIAID Data Ecosystem40

A Critical Role for the mTORC2 Pathway in Lung Fibrosis

纤维化肺病

mTOR信号通路

A characteristic of dysregulated wound healing in IPF is fibroblastic-mediated damage to lung epithelial cells within fibroblastic foci. In these foci, TGF-β and other growth factors activate fibrobla

NIAID Data Ecosystem20

Melatonin inhibits fibroblast cell functions and hypertrophic scar formation by enhancing autophagy through the MT2 receptor-inhibited PI3K/Akt /mTOR signaling. Melatonin inhibits fibroblast cell functions and hypertrophic scar formation by enhancing autophagy through the MT2 receptor-inhibited PI3K/Akt /mTOR signaling

肥厚性疤痕纤维化

mTOR信号通路

Hypertrophic scar (HS) is a fibrotic skin condition and characterized by abnormal proliferation of myofibroblasts and accumulation of extracellular matrix. Melatonin, an endogenous hormone, can allevi

NIAID Data Ecosystem10

Additional file 1 of CHKB-AS1 enhances proliferation and resistance to NVP-BEZ235 of renal cancer cells via regulating the phosphorylation of MAP4 and PI3K/AKT/mTOR signaling

mTOR信号通路

非编码RNA差异表达

Additional file 1. Differentially expressed lncRNAs.

Figshare2024-08-14 更新30

Targeting cancer heterogeneity by breaking a stem cell and epithelial niche interaction loop. Targeting cancer heterogeneity by breaking a stem cell and epithelial niche interaction loop

乳腺癌干细胞

肿瘤微环境

BMP7 promotes luminal INHBA production to expand mammary stem cell pool Overall design: We stimulated the HC11 cells using medium with or without BMP7 protein

NIAID Data Ecosystem10

Metastasis and Survival of Breast Cancer Stem Cells Mediated by Cytoskeleton Remodeling and PI3K/mTOR Signaling transcription factors

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