Oxidative damage and respiratory burst in Multiple Sclerosis
收藏官方服务:
资源简介:
Oxidative damage and respiratory burst in Multiple Sclerosis
应用场景:
创建时间:
2011-10-12
相关数据集
Mechanistic differences in neuropathic pain modalities revealed by correlating behavior with global expression profiling [microarray]
Chronic neuropathic pain is a major morbidity of neural injury, yet its mechanisms are incompletely understood. Hypersensitivity to previously non-noxious stimuli (allodynia) is a common symptom. Here
NIAID Data Ecosystem110
Cellular and extracellular tRNA-derived fragments demonstrate distinct signatures in cellular stress. Cellular and extracellular tRNA-derived fragments demonstrate distinct signatures in cellular stress
The cellular response to stress is an important determinant of disease pathogenesis. Uncovering the molecular fingerprints of distinct stress responses may yield novel biomarkers for different disease
NIAID Data Ecosystem50
Thirty-five proteins identified with altered levels of expression in response to transport stress (stressed) compared to non-stressed birds, as revealed by label-free MS/MS analysis using progenesis QI for proteomics.
Thirty-five proteins identified with altered levels of expression in response to transport stress (stressed) compared to non-stressed birds, as revealed by label-free MS/MS analysis using progenesis Q
NIAID Data Ecosystem20
Transcriptomic profiling in mice with CB1 deletion in primary sensory neurons
in this study, we investigated whether CB1R conditional knockout from peripheral sensory neurons (CB1cKO) in mice may present mechanical allodynia, thermal hyperalgesia and change transcriptome expres
NIAID Data Ecosystem70
The role of m6A/m-RNA methylation in stress response regulation
N6-Methyladenosine (m6A) and N6,2'-O-dimethyladenosine (m6Am) are abundant mRNA modifications that regulate transcript processing and translation. The role of both, here termed m6A/m, in the stress re
NIAID Data Ecosystem30



