MOESM4 of Exhaustion of mitochondrial and autophagic reserve may contribute to the development of LRRK2 G2019S -Parkinson’s disease
收藏Figshare2018-06-09 更新2026-04-08 收录
下载链接:
https://springernature.figshare.com/articles/MOESM4_of_Exhaustion_of_mitochondrial_and_autophagic_reserve_may_contribute_to_the_development_of_LRRK2___G2019S__-Parkinson_s_disease/6465353/1
下载链接
链接失效反馈官方服务:
资源简介:
Additional file 4: Table S2. Raw data of mitochondrial phenotype and autophagic print parameters. The following legend accompanies the table: Glu: glucose; Gal: galactose; NM-LRRK2 G2019S : Non-manifesting carriers of LRRK2 G2019S -mutation; PD- LRRK2 G2019S : patients with LRRK2 G2019S -mutation and clinically manifest PD; mtDNA: Mitochondrial DNA; mtRNA: Mitochondrial RNA; CI: Complex I; O2 consumption stimulated for CI: Oxygen consumption stimulated for Complex I substrates (pyruvate, malate and glutamate); CIV: Complex IV. Pa. P value when comparing NM-LRRK2 G2019S vs. controls; Pb. P value when comparing PD- LRRK2 G2019S vs. controls; Pc. P value when comparing NM-LRRK2 G2019S vs. PD-LRRK2 G2019S .
提供机构:
Mariona Guitart-Mampel; Glòria Garrabou
创建时间:
2018-06-09



