STAT6 acetylation in renal cancer metastasis

NIAID Data Ecosystem2026-03-13 收录

肾癌转移机制

蛋白质乙酰化靶点

数据链接：

https://www.ncbi.nlm.nih.gov/bioproject/PRJNA800658 数据链接链接失效反馈

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资源简介：

To discover the natural inhibitor recognizing STAT6 acetylation at K284 and block advanced renal cancer.

创建时间：

2022-01-26

Effects of HIF2α in renal cancer metastasis and sensitivity to repurposed drugs

肾癌转移机制

药物重定位

Background: in clear cell renal cell carcinoma (ccRCC), 80% of cases have biallelic inactivation of VHL gene, leading to constitutive activation of both HIF1α and HIF2α. As HIF2α is the driver of the

NIAID Data Ecosystem70

LncRNA from metastatic clear cell renal cell carcinoma (ccRCC) compared with non-metastatic clear cell renal carcinoma

肾癌转移机制

基因表达谱分析

A total of 3 metastatic and 3 non-metastatic ccRCC tissues were collected from patients. These tissues were flash frozen in liquid nitrogen immediately after surgery and subsequently stored at -80℃. A

NIAID Data Ecosystem50

Epigenetic expansion of VHL-HIF signal output drives multiorgan metastasis in renal cancer

肾癌转移机制

表观遗传调控

Inactivation of the von Hippel-Lindau tumor suppressor gene, VHL, is an archetypical tumor-initiating event in clear cell renal carcinoma (ccRCC) that leads to the activation of hypoxia-inducible tran

NIAID Data Ecosystem30

The SWI/SNF chromatin remodeling factor DPF3 regulates metastasis of ccRCC by modulating TGF-β signaling.

肾癌转移机制

TGF-β信号通路

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

NIAID Data Ecosystem60

RNA-sequencing of neutrophils derived from OS-RC-2-bearing mice

肾癌转移机制

粒细胞转录组

Advanced clear cell renal cell carcinoma (ccRCC) frequently causes systemic inflammation. Here, we identified the functional role and regulatory mechanism of inflammation driven by advanced ccRCC cell

NIAID Data Ecosystem40

STAT6 acetylation in renal cancer metastasis

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