Alternative splicing Hltf: intron retention-dependent activation of immune tolerance at the feto-maternal interface

NIAID Data Ecosystem2026-03-11 收录

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https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE114146

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This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

创建时间：

2019-03-19

Alternative splicing Hltf: intron retention-dependent activation of immune tolerance at the feto-maternal interface. Alternative splicing Hltf: intron retention-dependent activation of immune tolerance at the feto-maternal interface

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

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Alternative splicing Hltf: intron retention-dependent activation of immune tolerance at the feto-maternal interface (RNA-Seq). Alternative splicing Hltf: intron retention-dependent activation of immune tolerance at the feto-maternal interface (RNA-Seq)

Purpose: we tested the hypothesis that Hltf deletion in placenta either caused or exacerbated neonatal hypoglycemia via Hif-1α regulation of nutrient transporters. Methods: Individual samples [1 term

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Alternative splicing Hltf: intron retention-dependent activation of immune tolerance at the feto-maternal interface (RNA-Seq)

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Alternative splicing Hltf: intron retention-dependent activation of immune tolerance at the feto-maternal interface (Bisulfite-Seq). Alternative splicing Hltf: intron retention-dependent activation of immune tolerance at the feto-maternal interface (Bisulfite-Seq)

Purpose: Genome-wide methylation analysis was performed to determine whether deleting Hltf altered DNA methylation patterns, and whether differentially methylated regions (DMRs) coincided with changes

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Alternative splicing Hltf: intron retention-dependent activation of immune tolerance at the feto-maternal interface (Bisulfite-Seq)

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