TCRb-seq data from dermal T cells in SJL mice upon immunisation.

NIAID Data Ecosystem2026-03-11 收录

淋巴细胞受体谱分析

自身免疫性皮肤病

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https://www.ncbi.nlm.nih.gov/bioproject/PRJNA586880 数据链接链接失效反馈

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Comparison of TCRb clonotypes in scratch-induced skin wounds between autoimmune mice (EBA) and control mice

创建时间：

2019-10-31

DataSheet_1_Oxidative Stress-Mediated YAP Dysregulation Contributes to the Pathogenesis of Pemphigus Vulgaris.pdf

自身免疫性皮肤病

YAP蛋白

Pemphigus Vulgaris (PV) is a life-threatening autoimmune disease manifested with blisters in the skin and mucosa and caused by autoantibodies against adhesion protein desmoglein-3 (Dsg3) expressed in

NIAID Data Ecosystem30

Fiorentino 2006-7 dermatomyositis, lupus, control skin

自身免疫性皮肤病

基因表达谱分析

Background: Dermatomyositis (DM) is an autoimmune disease that mainly affects the skin, muscle, and lung. The pathogenesis of skin inflammation in DM is not well understood. Methodology and Findings:

NIAID Data Ecosystem20

Correlationa between antibody levels, eosinophil counts, and disease severity in untreated BP patients.

自身免疫性皮肤病

生物标志物关联分析

adetermined using Spearman’s rank correlation coefficient (r) and p = *breported as IU/ml, normal range ≤100.cexpressed as Index Units, positive test ≥19.dreported as BP index/BPDAI as described in th

Figshare2015-12-02 更新10

Table_2_C-type lectin receptor expression is a hallmark of neutrophils infiltrating the skin in epidermolysis bullosa acquisita.xlsx

自身免疫性皮肤病

粒细胞转录组

IntroductionInflammatory epidermolysis bullosa acquisita (EBA) is characterized by a neutrophilic response to anti-type VII collagen (COL7) antibodies resulting in the development of skin inflammation

NIAID Data Ecosystem20

HIF-1 regulates pathogenic cytotoxic T cells in lupus skin disease

自身免疫性皮肤病

空间转录组学

Human DLE demonstrates expression of HIF1- and cytotoxic molecules including granzyme B in the DLE lymphocytic infiltrate. Normalized gene expression per region of interest (ROI) in DLE or normal con

Mendeley Data2024-01-01 更新10

TCRb-seq data from dermal T cells in SJL mice upon immunisation.

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