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TOLLIP promotes GCRV infection by negatively modulating STING-mediated innate immunity

NIAID Data Ecosystem2026-05-10 收录

先天免疫调控

病毒致病机制

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE296862 数据链接链接失效反馈

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NOT PROVIDED; REQUESTED CIK CELLS; 2 repeated semples of each experinment

应用场景：

创建时间：

2025-09-24

病毒致病机制

蛋白质聚集分析

Disease-related prion protein (PrPSc), which is a structural isoform of the host-encoded cellular prion protein, is thought to be a causative agent of transmissible spongiform encephalopathies. Howeve

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TFEB and TFE3 regulate STING1-dependent immune responses by controlling type I interferon signaling

先天免疫调控

转录因子功能分析

STING1 is an essential component of the innate immune defense against a wide variety of pathogens. Whereas induction of Type I interferon (IFN) responses is one of the best-defined functions of STING1

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Crystal structure of the oligomerization domain of NSP4 from the rotavirus strain NCDV

病毒蛋白结构解析

病毒致病机制

Crystal structure of the oligomerization domain of NSP4 from the rotavirus strain NCDV

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Identification of a FOXO3/IRF7 circuit that limits inflammatory sequelae of antiviral responses (expression)

先天免疫调控

转录因子功能验证

We have used an unbiased systems approach to predict that a member of the forkhead family of transcription factors, FOXO3, is a negative regulator of a subset of antiviral genes. This prediction was v

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Data_Sheet_2_RNA-Seq Analysis Reveals the Role of Omp16 in Brucella-Infected RAW264.7 Cells.xls

细菌宿主互作

先天免疫调控

Brucellosis is an endemic zoonotic infectious disease in the majority of developing countries, which causes huge economic losses. As immunogenic and protective antigens at the surface of Brucella spp.

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TOLLIP promotes GCRV infection by negatively modulating STING-mediated innate immunity

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