Expression data from ischemia and reperfusion events in NRVMs using Coverslip Hypoxia
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Ischemia and Reperfusion Induce Differential Expression of Calpastatin and Its Homologue High Molecular Weight Calmodulin-Binding Protein in Murine Cardiomyocytes
In the heart, calpastatin (Calp) and its homologue high molecular weight calmodulin-binding protein (HMWCaMBP) regulate calpains (Calpn) by inhibition. A rise in intracellular myocardial Ca2+ during c
NIAID Data Ecosystem40
DataSheet4_Verapamil Alleviates Myocardial Ischemia/Reperfusion Injury by Attenuating Oxidative Stress via Activation of SIRT1.ZIP
Myocardial ischemia/reperfusion (I/R) injury is a potential complication of ischemic heart disease after recanalization. One of the primary reasons for I/R injury is the excessive accumulation of reac
NIAID Data Ecosystem10
STING aggravates ferroptosis-dependent myocardial ischemia-reperfusion injury by targeting GPX4 for autophagic degradation
To investigate the potential signal pathways affected by the deletion of the Sting gene in mice during ischemia-reperfusion (IR), we extracted the myocardial tissues of mice 24 hours after ischemia-re
NIAID Data Ecosystem10
Transition states (expression levels) during reprogramming using the five experimental recipes.
Transition states (expression levels) during reprogramming using the five experimental recipes.
Figshare2015-12-02 更新10
Table1_Somatostatin and Its Receptors in Myocardial Ischemia/Reperfusion Injury and Cardioprotection.xlsx
Little is known about the role of the neuropeptide somatostatin (SST) in myocardial ischemia/reperfusion injury and cardioprotection. Here, we investigated the direct cardiocytoprotective effect of SS
NIAID Data Ecosystem10
