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Supplemental Table 4 from Sensitizing Triple-Negative Breast Cancer to PI3K Inhibition by Cotargeting IGF1R

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数据链接：

https://figshare.com/articles/dataset/Supplemental_Table_4_from_Sensitizing_Triple-Negative_Breast_Cancer_to_PI3K_Inhibition_by_Cotargeting_IGF1R/22508065 数据链接链接失效反馈

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资源简介：

The correlation between Î"AUC and IGF1R mRNA and protein

应用场景：

创建时间：

2016-07-01

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Seventy-six FDA approved oncology drugs and emerging therapeutics were evaluated in 25 multiple myeloma (MM) and 15 non-Hodgkin’s lymphoma cell lines and in 113 primary MM samples. Ex vivo drug sensit

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Cytochalasin D as a potential MAPK family inhibitor for treating triple-negative breast cancer

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MAPK信号通路

Cytochalasin D, a potent fungal metabolite, has garnered considerable attention due to its diverse biological activities and potential pharmacological applications. Renowned for its ability to disrupt

Taylor & Francis Group2025-02-27 更新50

Human TNBC Cells 72h Treatment with 4.5nM OTSSP167 MELK Inhibitor vs. DMSO Control (SUM159). Homo sapiens

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MEK抑制剂机制

Microarray comparing TNBC cells treated for 72 hours with OTSSP167 MELK Inhibitor and control (DMSO) Overall design: Two-condition arrays: SUM159 cells were treated with either 4.5nM OTSSP167 MELK Inh

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Metadata record for the manuscript: Nivolumab in combination with cabozantinib for metastatic triple-negative breast cancer: A phase II and biomarker study

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癌症免疫生物标志物

Summary This metadata record provides details of the data supporting the claims of the related manuscript: “Nivolumab in combination with cabozantinib for metastatic triple-negative breast cancer:

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A novel orally available seleno-purine molecule suppresses triple-negative breast cancer cell proliferation and progression to metastasis by inducing cytostatic autophagy

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自噬调控

Patients with triple-negative breast cancer (TNBC) often have a poor prognosis largely due to lack of effective targeted therapy. Using a library of seleno-purines coupled to a high-throughput biochem

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Supplemental Table 4 from Sensitizing Triple-Negative Breast Cancer to PI3K Inhibition by Cotargeting IGF1R

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