The etiology of non-alcoholic steatohepatitis (NASH) has been complicated. An increasing body of literature has indicated that hepatic oxidative stress exerts a causal role in driving NASH. Neverthele
Background & Aims: Little is known about the molecular pathophysiology of non-alcoholic steatohepatitis (NASH). Methods: Total RNA from NASH livers and normal healthy livers were analyzed on a whole
Background & AimsPeriodontitis increases the risk of nonalcoholic fatty liver disease (NAFLD); however, the underlying mechanisms are unclear. Here, we show that gut dysbiosis induced by oral administ
