H3.3B controls aortic dissection progression by regulating vascular smooth muscle cells phenotypic transition and vascular inflammation.
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https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE229127
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To investigate the distribution of H3.3B in VSMC phenotypic transition, we infect VSMCs with shNT or shH3f3b virus. Chromatin immunoprecipitation DNA-sequencing (ChIP-seq) for histone variants H3.3 in primary mouse VSMCs with shNT or shH3f3b.
创建时间：
2024-10-12
