Multiomics study of NB4 cells treated with YW3-56

NIAID Data Ecosystem2026-05-02 收录

白血病耐药机制

多组学分子响应

数据链接：

https://www.ncbi.nlm.nih.gov/sra/SRP338024 数据链接链接失效反馈

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资源简介：

Genomic and proteome data of promyelocytic leukemia cells treated with PAD4 inhibitor YW3-56.

创建时间：

2024-10-01

Data_Sheet_1_Acute Myeloid Leukemia Chemo-Resistance Is Mediated by E-selectin Receptor CD162 in Bone Marrow Niches.PDF

白血病耐药机制

肿瘤微环境靶向治疗

The interactions of leukemia cells with the bone marrow (BM) microenvironment is critical for disease progression and resistance to treatment. We have recently found that the vascular adhesion molecul

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Targeting the MTF2-MDM2 Axis Sensitizes Refractory Acute Myeloid Leukemia to Chemotherapy [RNA-Seq]

白血病耐药机制

表观遗传靶向治疗

Deep sequencing has revealed that epigenetic modifiers are the most mutated genes in acute myeloid leukemia (AML). Thus, elucidating epigenetic dysregulation in AML is crucial to understand disease me

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Expression profile of shikonin resistant K562 cell line

白血病耐药机制

抗肿瘤药物作用机制

Expression profile of shikonin resistant K562 cell line

ChEBI2011-12-10 更新00

Supplementary Table S5 from Mitophagy Promotes Resistance to BH3 Mimetics in Acute Myeloid Leukemia

白血病耐药机制

基因编辑序列数据

Supplementary Table S5 shows sgRNA, primers, shRNA sequences used in this study.

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Additional file 6 of Single-cell transcriptomics reveals multiple chemoresistant properties in leukemic stem and progenitor cells in pediatric AML

白血病耐药机制

单细胞转录组学

Additional file 6: Table S5. 117 scRNA Differential Expressed Genes (DEGs) in HSC-like cells between resistant and sensitive groups. These genes met with the differential expression criteria (min.pct

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Multiomics study of NB4 cells treated with YW3-56

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