Tollip isoforms as key regulators of macrophage phenotype and response to LPS

ChEBI2010-09-18 更新2026-07-21 收录

巨噬细胞免疫调节

Tollip蛋白异构体功能

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Tollip isoforms as key regulators of macrophage phenotype and response to LPS

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2010-09-18

Mechanisms underlying divergent responses of genetically distinct macrophages to IL-4 [ATAC-seq]

巨噬细胞免疫调节

表观遗传变异

Mechanisms by which noncoding genetic variation influences gene expression remain only partially understood but are considered to be major determinants of phenotypic diversity and disease risk. Here,

NIAID Data Ecosystem80

Bmal1 regulates inflammatory response in macrophage

巨噬细胞免疫调节

表观遗传时序分析

Deletion of Arntl disrupted temporal inflammatory response in macrophage. Mechanistically, the acetylation status of lysine 27 of histone 3 (H3K27ac) was enhanced at the PU.1-containing enhancers in A

NIAID Data Ecosystem60

Additional file 1 of Identification of sulfur metabolism-related gene signature in osteoarthritis and TM9SF2’s sustenance effect on M2 macrophages' phagocytic activity

关节炎分子机制

巨噬细胞免疫调节

Additional file 1. Table 1. Gene lists for analysis.

NIAID Data Ecosystem40

Table 3_TMEM106A mediates atherosclerosis progression through macrophage-centered immune responses and chemokine signaling.xls

动脉粥样硬化机制

巨噬细胞免疫调节

BackgroundAtherosclerosis (AS) remains a leading cause of cardiovascular morbidity and mortality, characterized by intricate interactions between immune dysregulation and lipid metabolism abnormalitie

NIAID Data Ecosystem70

Gene expression profiling of human MSC-educated macrophages vs. classical macrophages from bone marrow and blood. Homo sapiens

巨噬细胞免疫调节

组织修复机制

In comparison to MØs, MEMs have increased expression of the inhibitory molecules PD-L1, PD-L2, in addition to markers of alternatively activated macrophages: CD206 and CD163. RNA-Seq analysis of MEMs,

NIAID Data Ecosystem50

Tollip isoforms as key regulators of macrophage phenotype and response to LPS

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