CFIm25 links alternative polyadenylation to glioblastoma tumour suppression
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CFIm25 links alternative polyadenylation to glioblastoma tumour suppression
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2014-05-15
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Additional file 4 of Testing for causality between systematically identified risk factors and glioma: a Mendelian randomization study
Additional file 4 Additional file, Table 1: Details of instrumented risk factors found from the systematic search of the literature. Additional file, Table 2: Power calculations for each instrumented
DataCite Commons2020-08-25 更新60
Additional file 2 of SRSF3 and SRSF7 modulate 3′UTR length through suppression or activation of proximal polyadenylation sites and regulation of CFIm levels
Additional file 2.
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To investigate the decay constants (half-lives) of transcript isoforms generated by alternative polyadenylation in proliferating and quiescent cells. To investigate the decay constants (half-lives) of transcript isoforms generated by alternative polyadenylation in proliferating and quiescent cells
Purpose: Alternative polyadenylation (APA) can result in the generation of transcripts that terminate at different locations within the gene, which can result in different 3' UTRs. 3´ UTRs are known t
NIAID Data Ecosystem20
Additional file 2 of Fused inverse-normal method for integrated differential expression analysis of RNA-seq data
Additional file 2. Full list of differentially expressed genes (DEGs) identified in scenario 1 by the inverse-normal (IN) method in glioblastoma (GBM) data application.
Figshare2022-08-06 更新50
Distinct, opposing functions for CFIm59 and CFIm68 in mRNA alternative polyadenylation of Pten and in the PI3K/Akt signalling cascade
The precise maintenance of PTEN dosage is crucial for tumor suppression across a wide variety of cancers. Post-transcriptional regulation of Pten heavily relies on regulatory elements encoded by its 3
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