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Data file S4 from Targeting STAT5 Signaling Overcomes Resistance to IDH Inhibitors in Acute Myeloid Leukemia through Suppression of Stemness

NIAID Data Ecosystem2026-04-30 收录

白血病耐药机制

STAT信号通路

数据链接：

https://figshare.com/articles/dataset/Data_file_S4_from_Targeting_STAT5_Signaling_Overcomes_Resistance_to_IDH_Inhibitors_in_Acute_Myeloid_Leukemia_through_Suppression_of_Stemness/30699343 数据链接链接失效反馈

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STAT5 consensus binding motif around HOX genes

应用场景：

创建时间：

2022-12-02

白血病耐药机制

RNAm6A修饰分析

N6-Methyladenosine (m6A) is the most abundant post-transcriptional modification in eukaryotes, the imbalance of which is reported to be associated with various pathological processes, including drug r

NIAID Data Ecosystem60

Mapping the glucocorticoid gene regulatory network and alterations that contribute to steroid resistance in childhood acute lymphoblastic leukemia [Hi-ChIP]. Mapping the glucocorticoid gene regulatory network and alterations that contribute to steroid resistance in childhood acute lymphoblastic leukemia [Hi-ChIP]

白血病耐药机制

基因调控网络

Glucocorticoids (GCs) are a mainstay of contemporary, multi-drug chemotherapy in the treatment of acute lymphoblastic leukemia (ALL). Although overall survival rates of childhood ALL have improved, re

NIAID Data Ecosystem30

Correlations between constitutive p-STAT5, TPO induced p-STAT5, and IL6 induced p-STAT3 MFI values and disease parameters tested in circulating CD34 + cells of CALR + patients with PMF.

原发性骨髓纤维化

STAT信号通路

Correlations between constitutive p-STAT5, TPO induced p-STAT5, and IL6 induced p-STAT3 MFI values and disease parameters tested in circulating CD34+ cells of CALR+ patients with PMF.

NIAID Data Ecosystem30

DDX6 undergoes phase separation to modulate metabolic plasticity and drug resistance in AML [CRISPR Screen]

白血病耐药机制

CRISPRi筛选

Chemoresistance in acute myeloid leukemia (AML) is a major cause of poor prognosis and frequent relapse among patients. Under stress conditions such as chemotherapy or radiotherapy, processing bodies

NIAID Data Ecosystem30

Growth Factor and Th2 Cytokine Signaling Pathways Converge at STAT6 to Promote Arginase Expression in Progressive Experimental Visceral Leishmaniasis

内脏利什曼病风险分析

STAT信号通路

Host arginase 1 (arg1) expression is a significant contributor to the pathogenesis of progressive visceral leishmaniasis (VL), a neglected tropical disease caused by the intracellular protozoan Leishm

NIAID Data Ecosystem30

Data file S4 from Targeting STAT5 Signaling Overcomes Resistance to IDH Inhibitors in Acute Myeloid Leukemia through Suppression of Stemness

资源简介：

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