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Additional file 1: of Transcriptomic analysis of immune response to bacterial lipopolysaccharide in zebra finch (Taeniopygia guttata)

Figshare2019-08-14 更新2026-04-29 收录

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炎症基因表达

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https://figshare.com/articles/dataset/Additional_file_1_of_Transcriptomic_analysis_of_immune_response_to_bacterial_lipopolysaccharide_in_zebra_finch_Taeniopygia_guttata_/9631343 数据链接链接失效反馈

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Upregulated DEGs in hypothalamus. Raw DEG data for upregulated genes in hypothalamus. (XLSX 344 kb)

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创建时间：

2019-08-14

炎症基因表达

SWI/SNF染色质重塑

As one of the core components of the SWI/SNF complex, SMARCC1 (SAF155, SRG3) plays essential role in activation of the late inflammatory genes in response to microbial challenge. However, little is kn

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Inflammatory program of CF-TG cells transfected with cftr gene.

囊性纤维化基因治疗

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The expression levels of seven commonly regulated genes were determined by qRT-PCR. The results are expressed as the fold changes between CF-TG cells and wt TG cells, and between CF-TG cells transfect

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Targeted transcriptomic analysis of C57BL/6 and BALB/c mice during progressive chronic Toxoplasma gondii infection reveals changes in host and parasitic gene expression relating to neuropathology and resolution [Neuroinflammation]. Targeted transcriptomic analysis of C57BL/6 and BALB/c mice during progressive chronic Toxoplasma gondii infection reveals changes in host and parasitic gene expression relating to neuropathology and resolution [Neuroinflammation]

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Toxoplasma gondii is a resilient parasite that infects a multitude of warm-blooded hosts and results in a lifelong chronic infection requiring continuous responses by the host. Chronic infection is ch

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Distinct molecular signatures of mild extrinsic and intrinsic atopic dermatitis. Homo sapiens

特应性皮炎分子机制

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Atopic dermatitis (AD) is a common inflammatory skin disease with underlying defects in epidermal function and immune responses. The goal of this study was to investigate differences in gene expressio

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A stimulus-contingent positive feedback loop enables IFN-β dose-dependent activation of pro-inflammatory genes. A stimulus-contingent positive feedback loop enables IFN-β dose-dependent activation of pro-inflammatory genes

干扰素信号通路

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Type I interferons (IFN) induce powerful anti-viral and innate immune responses via the transcription factor, IFN-stimulated gene factor (ISGF3). However, in some pathological contexts type I IFNs are

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Additional file 1: of Transcriptomic analysis of immune response to bacterial lipopolysaccharide in zebra finch (Taeniopygia guttata)

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