AR cooperates with SMAD4 to maintain muscle homeostasis
收藏NIAID Data Ecosystem2026-03-13 收录
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https://www.ncbi.nlm.nih.gov/sra/SRP341585
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资源简介：
The goal of this study was to determine the mechanism of androgne in driving muscle hypertrophy programmes. CONCLUSION: AR transcriptionally cooperates with SMAD4 and this affect is lost in the presence of expansion of the polyglutamine repeat in the AR gene. Overall design: RNA-seq was performed in quadricpes femoris from SBMA (spinal bulbar muscular atrophy) AR100Q mice treated with AAV vectors expressing BMP7 or GFP mock control. Untreated wild-type mice (C57BL/6) were analysed in parallel as additional controls.
创建时间：
2022-05-29
