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资源简介：
MYCN regulated miRNAs
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创建时间：
2015-07-24
相关数据集
Ubiquitin-dependent transfer of PAF1c from MYC onto RNA Polymerase controls double-strand break repair at active promoters [ChIP-seq BI8626]. Ubiquitin-dependent transfer of PAF1c from MYC onto RNA Polymerase controls double-strand break repair at active promoters [ChIP-seq BI8626]
MYC family proteins are oncogenic transcription factors that can globally affect the function of RNA Polymerase II (RNAPII). The ability of MYC proteins to promote transcription elongation depends on
NIAID Data Ecosystem50
Mediator Subunit 30 Directs Myc Oncogenesis [PRO-seq]. Mediator Subunit 30 Directs Myc Oncogenesis [PRO-seq]
Mediator Subunit 30 Directs Myc Oncogenesis [PRO-seq] Overall design: To investage the effect of med30 overespression on transcription, PRO-seq was performed in teton MED30 overexpression Mia PaCa-2 c
NIAID Data Ecosystem20
ScanFold2 output files for MYC intron 2 from selected species.
ScanFold2 output files for MYC intron 2 from selected species.
NIAID Data Ecosystem10
SIRT7 suppresses tumorigenesis by attenuating MYC activity
SIRT7 is a member of the mammalian sirtuin family of NAD+ dependent deacylases, and interacts with RNA polymerase I and UBF to regulate rDNA transcription. Various studies in mammalian cells and hum
NIAID Data Ecosystem30
MYC threshold required to maintain tumor phenotype
We demonstrate that there is a level of MYC expression that is required for maintaining a tumor phenotype and at this threshold there is a switch from a state of cellular proliferation to a state of
NIAID Data Ecosystem20
