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Transcription profiling of mouse hairless mutant Hos: HR-1 to investigate mechanism alopecia

ChEBI2008-10-18 更新2026-07-21 收录

小鼠毛发模型研究

脱发分子机制

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-6866 数据链接链接失效反馈

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资源简介：

Transcription profiling of mouse hairless mutant Hos: HR-1 to investigate mechanism alopecia

应用场景：

创建时间：

2008-10-18

脱发分子机制

毛囊细胞信号通路

Our data showed that miR-221 had a profound effect on the biological function of dermal papilla cells (DPCs) and dermal sheath cells (DSCs) of hair follicle. Overexpression of miR-221 affected the MAP

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NFI Transcription Factors Govern Stem Cell Chromatin Accessibility and Guard Against Irreversible Hair Loss. NFI Transcription Factors Govern Stem Cell Chromatin Accessibility and Guard Against Irreversible Hair Loss

细胞染色质调控

脱发分子机制

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

NIAID Data Ecosystem30

Ox40-Cre-mediated deletion of BRD4 reveals an unexpected phenotype of hair follicle stem cells in alopecia

毛囊干细胞

脱发分子机制

To identify the mechanisms of impaired hair regeneration in Brd4-deleted follicle stem cells, we FACS sorted HFSCs from Rag1-/-Ox40-Cre and Rag1-/-Brd4f/fOx40-Cre mice and performed RNA-seq analysis c

NIAID Data Ecosystem20

Transcriptomic analysis of frontal fibrosing alopecia

皮肤癌转录组学

脱发分子机制

We undertook transcriptome profiling with RNA-sequencing of scalp skin from seven cases of European ancestry and seven matched controls. Transcriptomic analysis was undertaken in 7 cases vs 7 matched

NIAID Data Ecosystem60

Ox40-Cre-mediated deletion of BRD4 reveals an unexpected phenotype of hair follicle stem cells in alopecia

毛囊干细胞

脱发分子机制

NIAID Data Ecosystem20