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UNC50 prompts G1/S transition and proliferation in HCC through regulation of epidermal growth factor receptor trafficking

ChEBI2014-11-17 更新2026-07-21 收录

肝细胞癌机制研究

EGF信号传导

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-63322 数据链接链接失效反馈

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UNC50 prompts G1/S transition and proliferation in HCC through regulation of epidermal growth factor receptor trafficking

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2014-11-17

神经科学

EGF信号传导

The late onset of neurodegeneration in humans indicates that the survival and function of cells in the nervous system must be maintained throughout adulthood. In the optic lamina of the adult Drosophi

NIAID Data Ecosystem50

DataSheet1_Metabolomics profiling of AKT/c-Met-induced hepatocellular carcinogenesis and the inhibitory effect of Cucurbitacin B in mice.docx

肝细胞癌机制研究

抗肿瘤药物代谢组学

Hepatocellular carcinoma (HCC), the most common kind of liver cancer, accounts for the majority of liver cancer diagnoses and fatalities. Clinical aggressiveness, resistance to traditional therapy, an

NIAID Data Ecosystem40

hepatocellular carcinoma vs. corresponding noncancerous liver or normal liver

肝细胞癌机制研究

抗癌靶点发现

Hepatocellular carcinoma (HCC) remains a major health problem worldwide, and HCC patients have a poor prognostic outcome. In this study, we systematically disclose mechanisms of hepatocarcinogenesis,

NIAID Data Ecosystem20

The Akt-SRPK-SR axis constitutes a major pathway in transducing EGF signaling to regulate alternative splicing in the nucleus. Homo sapiens

EGF信号传导

剪接机制调控

Pre-mRNA splicing is regulated by developmental and environmental cues, but little is known about how specific signals are transduced in mammalian cells to regulate this critical gene expression step.

NIAID Data Ecosystem40

Table1_ARPC2: A Pan-Cancer Prognostic and Immunological Biomarker That Promotes Hepatocellular Carcinoma Cell Proliferation and Invasion.DOCX

肿瘤生物标志物

肝细胞癌机制研究

Background: Actin-related protein 2/3 complex subunit 2 (ARPC2) plays a fundamental role in actin filament nucleation and is critical for tumor cell migration and invasion. However, its abnormal expre

NIAID Data Ecosystem20

UNC50 prompts G1/S transition and proliferation in HCC through regulation of epidermal growth factor receptor trafficking

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