Chemical Inhibition of the RORγt-dependent Transcriptional Network in Th17 cells
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Chemical Inhibition of the RORγt-dependent Transcriptional Network in Th17 cells
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2014-04-17
相关数据集
Genome-wide detection of STAT3 binding sites in human Th17 cells. Homo sapiens
STAT3 is a major transcription factor driving the polarization of Th17 cells in response to IL-6, TGF-β and IL1-β. STAT3 is phosphorylated and forms a homodimer and translocates into the nucleus. Ther
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Transcription factor TCF1 binds Rorgt and orchestrates a regulatory network that determines homeostatic Th17 cell state [ATAC-seq]. Transcription factor TCF1 binds Rorgt and orchestrates a regulatory network that determines homeostatic Th17 cell state [ATAC-seq]
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Table_2_Interleukin 27 is a novel cytokine with anti-inflammatory effects against spondyloarthritis through the suppression of Th17 responses.xlsx
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Genome-wide binding of STAT3 and STAT5 under Th17 conditions (ChIP-Seq)
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