Loss of ULK1 Promotes TLR4 Acetylation in Bone Marrow-Derived Macrophages

Name: Loss of ULK1 Promotes TLR4 Acetylation in Bone Marrow-Derived Macrophages
Creator: Sun Yat-sen University
Published: 2026-06-01T14:27:35+08:00

integrated proteome resources2026-06-12 收录

先天免疫信号通路

巨噬细胞调控

数据链接：

https://www.iprox.cn/page/project.html?id=IPX0017365000 数据链接链接失效反馈

官方服务：

资源简介：

Loss of ULK1 Promotes TLR4 Acetylation in Bone Marrow-Derived Macrophages

提供机构：

Sun Yat-sen University

创建时间：

2026-06-01

Mechanisms Establishing TLR4-Responsive Activation States of Inflammatory Response Genes

先天免疫信号通路

炎症与表观遗传调控

Precise control of the innate immune response is required for resistance to microbial infections and maintenance of normal tissue homeostasis. Because this response involves coordinate regulation of h

NIAID Data Ecosystem60

Structure of apo-SPO1 Tad2

蛋白质结构解析

先天免疫信号通路

Structure of apo-SPO1 Tad2 Descriptor: Gp34.65 Authors: Xiao, Y, Feng, Y. Deposit date: 2023-09-25 Release date: 2024-09-25 Last modified: 2024-12-04 Method: X-RAY DIFFRACTION (2.27 Å) Cite: Single ph

Protein Data Bank Japan2024-12-04 更新50

Host-pathogen dynamics through secretome analysis of stimulated macrophages

先天免疫信号通路

病原体-宿主相互作用

The Toll-like receptors (TLRs) recognize different pathogen associated molecular patterns (PAMPs) and promote MyD88 dependent and independent responses. Previously, we have reported the discovery of t

NIAID Data Ecosystem40

Transcriptional affects of KLF2 deletion in bone marrow-derived macrophages

巨噬细胞调控

转录组学分析

We report bulk RNA-sequencing of bone marrow-derived macrophages (BMDMs) harvested from myeloid-specific KLF2 knockout (K2KO) and LysMCre control (LysM) mice Overall design: BMDMs were isolated from 3

NIAID Data Ecosystem60

Supplemental data for the manuscript "Effects of a CCR2 antagonist on macrophages and Toll-like receptor 9 expression in a mouse model of diabetic nephropathy"

糖尿病机制

巨噬细胞调控

We classified kidney macrophages (Mφs) into bone marrow-derived (BM-) macrophages expressing high CD11b and tissue-specific resident (Res-) macrophages expressing low CD11b. In diabetic nephropathy (D

Figshare2021-11-10 更新40

Loss of ULK1 Promotes TLR4 Acetylation in Bone Marrow-Derived Macrophages

资源简介：

相关数据集