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A timecourse analysis of the genetic extinction of NRAS in an inducible mouse model of melanoma.

ChEBI2012-08-08 更新2026-07-21 收录

黑色素瘤分子机制

基因功能时间序列分析

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-39985 数据链接链接失效反馈

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资源简介：

A timecourse analysis of the genetic extinction of NRAS in an inducible mouse model of melanoma.

应用场景：

创建时间：

2012-08-08

黑色素瘤分子机制

基因功能注释

Significant GO terms for the top 300 genes that distinguish primary from cutaneous melanoma.

NIAID Data Ecosystem70

Importance of RAD6B in activation of canonical WNT signaling in melanoma development

黑色素瘤分子机制

Wnt信号通路调控

We have demonstrated that RAD6B, a ubiquitin conjugating enzyme that is a principal component of translesion synthesis pathway, stabilizes and transcriptionally activates beta-catenin by lysine 63-lin

NIAID Data Ecosystem60

Transcription profiling by array of human SK-MEL-2 cells infected with Ad-E2F-1 after treatment with doxorubicin

黑色素瘤分子机制

化疗联合治疗

SK-MEL-2 cells treated with Ad-E2F-1 (MOI 2), Ad-E2F-1 (MOI 2)plus doxorubicin 0.1uM, or Ad-LacZ (MOI 2) plus doxorubicin 0.1uM. The combination of E2F-1 gene therapy and chemotherapy produces a syner

NIAID Data Ecosystem50

Additional file 12: of Loss of miR-204 expression is a key event in melanoma

黑色素瘤分子机制

小RNA功能分析

Table S7. The mutations and miR inhibition for each of the cell lines used in the in vitro experiments. (XLSX 9Â kb)

NIAID Data Ecosystem60

m6A IP sequencing analysis of m6A mRNA methylation regulation by FTO in melanoma cells. m6A IP sequencing analysis of m6A mRNA methylation regulation by FTO in melanoma cells

RNA表观遗传学

黑色素瘤分子机制

N6-methyladenosine (m6A) RNA methylation is the most abundant internal chemical modifications in eukaryotic messenger RNA (mRNA) as well as long non-coding RNA (lncRNA). Recently, m6A RNA methylation

NIAID Data Ecosystem50

A timecourse analysis of the genetic extinction of NRAS in an inducible mouse model of melanoma.

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