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MuRF1 regulation of Fenofibrateâs induction of spontaneous cardiac hypertrophy in vivo.

ChEBI2016-03-01 更新2026-07-12 收录

心脏肥大机制

药物诱导毒性

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-68480 数据链接链接失效反馈

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资源简介：

MuRF1 regulation of Fenofibrateâs induction of spontaneous cardiac hypertrophy in vivo.

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创建时间：

2016-03-01

心脏肥大机制

心肌细胞生长调控

Background: Cardiac hypertrophy, defined as a stress-induced increase in heart mass/size, is a major risk factor for adverse cardiovascular events including heart failure and arrhythmia. Within this g

NIAID Data Ecosystem80

Physiologic cardiac hypertrophy

心脏肥大机制

运动生理学

Dahl salt-sensitive (DS) rats were obtained from Harlan Sprague Dawley Laboratory at 5 weeks of age. At 6 weeks of age, physiologic cardiac hypertrophy was generated by a vigorous daily exercise regim

NIAID Data Ecosystem70

Deubiquitinase JOSD2 improves calcium handling and attenuates cardiac hypertrophy and dysfunction through stabilizing SERCA2a in cardiomyocytes

心脏肥大机制

去泛素化酶

Construction of transcriptome sequencing library and transcriptome sequencing was completed by LC-Bio Technology Co., Ltd. (Hangzhou, China). The expression profile of Machado-joseph deubiquitinases (

NIAID Data Ecosystem10

The role of Clk3 in the development of heart hypertrophy. The role of Clk3 in the development of heart hypertrophy

心脏肥大机制

RNA测序

We used RNA-seq technology to characterise the extent of transcriptional response to transverse aortic compression (TAC) in mouse models, both in wild type and in Clk3-knocked down animals. We thereby

NIAID Data Ecosystem80

Expression data from mouse heart with and without GATA4 S105A mutation; with and without adrenergic stress. Mus musculus

心脏肥大机制

基因表达调控

Serine 105 phosphorylation of GATA4 is necessary for stress-induced cardiac hypertrophy in vivo. We used microarrays to detail global gene expression program alterations that arise in response to the

NIAID Data Ecosystem60

MuRF1 regulation of Fenofibrateâs induction of spontaneous cardiac hypertrophy in vivo.

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MuRF1 regulation of Fenofibrateâs induction of spontaneous cardiac hypertrophy in vivo.