Setdb1 ChIP-seq
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下载链接:
https://www.ncbi.nlm.nih.gov/bioproject/PRJNA890871
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Setdb1 ChIP-seq
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创建时间:
2022-10-15
相关数据集
SETDB1 safeguards genome stability against necroptosis and bowel inflammation (ChIP-seq)
The aetiology of bowel inflammatory disease (IBD) is a multifactorial interplay between heredity and environment1-3. Here we report SETDB1, a histone methyltransferase (HMTs) for histone H3 lysine 9 t
NIAID Data Ecosystem10
SETDB1 safeguards genome stability against necroptosis and bowel inflammation (ChIP-seq)
The aetiology of bowel inflammatory disease (IBD) is a multifactorial interplay between heredity and environment1-3. Here we report SETDB1, a histone methyltransferase (HMTs) for histone H3 lysine 9 t
NIAID Data Ecosystem10
SETDB1 safeguards genome stability against necroptosis and bowel inflammation (ChIP-seq). SETDB1 safeguards genome stability against necroptosis and bowel inflammation (ChIP-seq)
The aetiology of bowel inflammatory disease (IBD) is a multifactorial interplay between heredity and environment1-3. Here we report SETDB1, a histone methyltransferase (HMTs) for histone H3 lysine 9 t
NIAID Data Ecosystem20
A somatic role for the histone methyltransferase Setdb1 in endogenous retrovirus silencing [ChIP-Seq]
Subsets of endogenous retroviruses (ERVs) are derepressed in mouse embryonic stem cells (mESCs) deficient for Setdb1, which catalyzes histone H3 lysine 9 trimethylation (H3K9me3). Most of those ERVs,
NIAID Data Ecosystem00



