ChIP-seq Identification of Weakly Conserved Heart Enhancers
收藏官方服务：
资源简介：
ChIP-seq Identification of Weakly Conserved Heart Enhancers
应用场景：
创建时间：
2010-08-22
相关数据集
Control ChIP-seq from activated B cell (ENCSR003FJP)
For data usage terms and conditions, please refer to http://www.genome.gov/27528022 and http://www.genome.gov/Pages/Research/ENCODE/ENCODE_Data_Use_Policy_for_External_Users_03-07-14.pdf https://www.e
NIAID Data Ecosystem50
TF ChIP-seq from whole organism (ENCSR861RPB)
ChIP-seq on transgenic flies expressing CG13775-eGFP fusion proteins. The IP was performed using an anti-GFP antibody. For data usage terms and conditions, please refer to http://www.genome.gov/27528
NIAID Data Ecosystem20
ZXDA/B and CENP-A/B Chromatin immunoprecipitation sequencing (ChIP-seq) in human colon carcinoma cells.. ZXDA/B and CENP-A/B Chromatin immunoprecipitation sequencing (ChIP-seq) in human colon carcinoma cells.
The goal of this experiment was to investigate the role of the FBXO38 ubiquitin ligase in the regulation of centromeric proteins deposition in HCT116 cell line.
NIAID Data Ecosystem10
Control ChIP-seq from K562 (ENCSR956GFH)
K562 immortalized cell line stably expressing C-terminal eGFP-ZNF778 fusion protein. For data usage terms and conditions, please refer to http://www.genome.gov/27528022 and http://www.genome.gov/Page
NIAID Data Ecosystem30
MLL3 Loss Drives Metastasis by Promoting a Hybrid Epithelial-Mesenchymal Transition State [ChIP-seq UTX and MLL4]. MLL3 Loss Drives Metastasis by Promoting a Hybrid Epithelial-Mesenchymal Transition State [ChIP-seq UTX and MLL4]
Phenotypic plasticity associated with the hybrid epithelial-mesenchymal transition (EMT) state is crucial to metastatic seeding and outgrowth. However, the mechanisms controlling the induction of hybr
NIAID Data Ecosystem10
