This Series reports data from a CTCF ChIP-Seq experiment performed in F1-hybrid mouse trophoblast stem cells (TSCs). The data are part of a larger study examining inactive X gene expression and chroma
Cell fate decisions are closely associated with changes in gene expression programs. A large number of post-translational modifications of core histones contribute to controlling the expression of gen
Our studies have revealed that a large class of CTCF binding sites – namely the upstream sites – conform neither to a conformational nor a local recombinase activating role. Their conserved spatial di
We sequenced CTCF binding sites in Akt1+/+ or Akt1-/- mouse embryonic fibroblast (MEF) cell line. Overall design: The information of CTCF binding sites in wildtype (WT) and Akt1-/- MEF cells was gener
ChIPmentation-seq analysis of CTCF and RAD21 in wildtype and mutant CTCF binding sites alleles ChIPmentation-seq analysis of CTCF and RAD21 in wildtype and mutant CTCF binding sites alleles
