G3BP2 in Oscillatory Shear Stress-induced Atherosclerosis

NIAID Data Ecosystem2026-03-12 收录

动脉粥样硬化机制

生物力学分子病理

数据链接：

https://www.ncbi.nlm.nih.gov/bioproject/PRJNA706049 数据链接链接失效反馈

官方服务：

资源简介：

To illucidate the role of G3BP2 in the pathogenesis of atherosclerosis formation under oscillatory shear stress

创建时间：

2021-03-02

The influence of nt-LNA on cell composition and phenotypees in the atherosclerotic lesion.

动脉粥样硬化机制

单细胞RNA测序

Raw fastq files from paired-end, Illumina-based sequencing on NovaSeq6000 Apoe-/- mice were fed a western diet for 4 weeks and administered saline or nt-LNA (30 mg/kg) weekly. After 4 weeks, aortic a

NIAID Data Ecosystem80

Effects of acLDL loading on macrophage. Mus musculus

泡沫细胞模型

动脉粥样硬化机制

acLDL loading of mouse peritoneal macrophage is an in vitro foam cell model. We have used microarray analysis and proteomics to characterize the effects of acLDL loading on the cells. Overall design:

NIAID Data Ecosystem50

LncRNA ICR regulates endothelium inflammation through feedback activation of NF-κB signaling

动脉粥样硬化机制

血管炎症调控

The responsive and abundant lncRNAs to pro-inflammatory factors in human ECs were screened and validated by RNA sequencing and qPCR. ICAM1-related non-coding RNA (ICR) was identified as the most poten

NIAID Data Ecosystem40

Activation of the pluripotency factor OCT4 in smooth muscle cells is atheroprotective. doi: 10.1038/nm.4109

动脉粥样硬化机制

平滑肌细胞转录组

The multiple claims about reactivation of the embryonic stem cell (ESC) pluripotency factor OCT4 in somatic cells are highly controversial due to the fact that there is no direct evidence that OCT4 ha

NIAID Data Ecosystem70

Nicotinic Acid Receptor GPR109A Is Down-Regulated in Human Macrophage-Derived Foam Cells

动脉粥样硬化机制

泡沫细胞

Nicotinic acid (NA) regresses atherosclerosis in human imaging studies and reduces atherosclerosis in mice, mediated by myeloid cells, independent of lipoproteins. Since GPR109A is expressed by human

NIAID Data Ecosystem40

G3BP2 in Oscillatory Shear Stress-induced Atherosclerosis

资源简介：

相关数据集