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MOESM2 of BMP3 suppresses colon tumorigenesis via ActRIIB/SMAD2-dependent and TAK1/JNK signaling pathways

NIAID Data Ecosystem2026-03-11 收录

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https://figshare.com/articles/dataset/MOESM2_of_BMP3_suppresses_colon_tumorigenesis_via_ActRIIB_SMAD2-dependent_and_TAK1_JNK_signaling_pathways/10086305 数据链接链接失效反馈

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Additional file 2. GO and KEGG pathway analysis.

应用场景：

创建时间：

2019-10-28

胚胎干细胞分化

JNK信号通路

The capacity of embryonic stem cells to differentiate into all lineages of mature organism is precisely regulated by cellular signaling factors. STAT3 is a crucial transcription factor that plays a ce

NIAID Data Ecosystem50

dMyc is required in retinal progenitors to prevent JNK-mediated retinal glial activation

视网膜发育

JNK信号通路

In the nervous system, glial cells provide crucial insulation and trophic support to neurons and are important for neuronal survival. In reaction to a wide variety of insults, glial cells respond with

Figshare2017-03-21 更新30

Interaction of Hematopoietic Progenitor Kinase 1 with Adapter Proteins Crk and CrkL Leads to Synergistic Activation of c-Jun N-Terminal Kinase

JNK信号通路

细胞激活机制

Hematopoietic progenitor kinase 1 (HPK1), a mammalian Ste20-related protein kinase, is an upstream activator of c-Jun N-terminal kinase (JNK). In order to further characterize the HPK1-mediated JNK si

PubMed Central10

Table1_Creatinine accelerates APAP-induced liver damage by increasing oxidative stress through ROS/JNK signaling pathway.docx

肝损伤机制

JNK信号通路

Serum creatinine is an endogenous biomarker to estimate glomerular filtration rate (GFR) and is commonly used to assess renal function in clinical practice. Acetaminophen (APAP), the most available an

NIAID Data Ecosystem30

WP5434 - Cancer pathways - Homo sapiens

癌症信号通路

生物信息学通路分析

This pathway is based on [Pathways in Cancer](https://www.genome.jp/pathway/hsa05200) from KEGG. It represents a combination of pathways affected in various cancers. Interactions depicted in orange ar

NIAID Data Ecosystem10

MOESM2 of BMP3 suppresses colon tumorigenesis via ActRIIB/SMAD2-dependent and TAK1/JNK signaling pathways

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