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miR-196a is able to restore the aggressive phenotype of Annexin A1 CRISPR/Cas9 knock-out pancreatic cancer cells

ChEBI2018-08-31 更新2026-07-12 收录

胰腺癌机制

CRISPR/Cas9基因编辑

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-6599 数据链接链接失效反馈

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资源简介：

miR-196a is able to restore the aggressive phenotype of Annexin A1 CRISPR/Cas9 knock-out pancreatic cancer cells

应用场景：

创建时间：

2018-08-31

胰腺癌机制

肿瘤微环境

ObjectivePancreatic ductal adenocarcinoma (PDAC) is a highly malignant neoplasm with rising incidence worldwide. Gremlin 1 (GREM1), a regulator of bone morphogenetic protein (BMP) signaling, fine-tune

NIAID Data Ecosystem80

Neural invasion induces cachexia via astrocytic activation of neural route in pancreatic cancer

胰腺癌机制

神经侵袭机制

Pancreatic cancer is characterized by a high frequency of cachexia, pain and neural invasion (N-inv). Neural damage is occurred by N-inv and modulates pain and muscle atrophy via the activation of ast

NIAID Data Ecosystem60

Analysis of conditioned medium cytokine levels of pancreatic cancer cells after JAG1 RNAi

胰腺癌机制

JAG1

To identify cytokines secreted by pancreatic cancer cells, we compared the cytokine profiles of conditioned medium from pancreatic cancer cell Su.86.86 treated with or without JAG1 siRNA, using a Huma

NIAID Data Ecosystem40

Fisetin induces autophagy in pancreatic cancer cells via endoplasmic reticulum stress- and mitochondrial stress-dependent pathways. Fisetin induces autophagy in pancreatic cancer cells via endoplasmic reticulum stress- and mitochondrial stress-dependent pathways

胰腺癌机制

自噬通路分析

Pancreatic cancer is one of the most aggressive tumors and has poor survival rates. Fisetin, a natural flavonoid, was recently reported to have antitumor effects in various cancer models. Autophagy is

NIAID Data Ecosystem60

Application of non-invasive low-intensity pulsed electric field with thermal cycling-hyperthermia for synergistically enhanced anticancer effect of chlorogenic acid on PANC-1 cells

胰腺癌机制

电热联合疗法

Most existing cancer treatments involve high-cost chemotherapy and radiotherapy, with major side effects, prompting effort to develop alternative treatment modalities. It was reported that the combina

NIAID Data Ecosystem50

miR-196a is able to restore the aggressive phenotype of Annexin A1 CRISPR/Cas9 knock-out pancreatic cancer cells

资源简介：

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