E-MEXP-3761

NIAID Data Ecosystem2026-05-02 收录

肌肉疾病分子机制

细胞因子刺激转录组学

数据链接：

https://www.omicsdi.org/dataset/atlas-experiments/E-MEXP-3761 数据链接链接失效反馈

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资源简介：

Transcription profiling by array of human myotubues derived from three patients carrying mutations in LPIN1 gene and three matched controls with or without TNF-alpha/IL-1-beta treatment

创建时间：

2025-01-22

Gene expression profiles using microarrays in mdx and mdx/MMP-2-/- mice at 1 and 3 months of age. Mus musculus

肌营养不良模型

肌肉疾病分子机制

Matrix metalloprotease (MMP) -2 has been reported to be up-regulated in skeletal muscle in the lethal X-linked muscle disorder Duchenne muscular dystrophy (DMD), which is caused by loss of dystrophin.

NIAID Data Ecosystem70

Temporal expression profiling of beta- and gamma-sarcoglycan deficient mice. Mus musculus

肌肉疾病分子机制

时序基因表达分析

Temporal expression profiling of hindlimb muscle of beta- and gamma-sarcoglycan deficient mice aged 1, 2.5, 4, 6, 8, 10, 12, 14 and 20 weeks. Keywords: time course Overall design: 4 closed loop-design

NIAID Data Ecosystem40

Canine skeletal muscle RNAscope raw data and analysis

肌肉疾病分子机制

肌营养不良蛋白

Raw image data, imageJ macros used for analysis, and analysis of healthy and dystrophic canine muscle probed with dystrophin multiplex ISH RNAscope probes. Data used for manuscriptSingle-transcript mu

NIAID Data Ecosystem40

Increased Systemic HSP70B Levels in Spinal Muscular Atrophy Infants. Increased Systemic HSP70B Levels in Spinal Muscular Atrophy Infants

脊髓性肌萎缩症生物标志物

肌肉疾病分子机制

RNA sequencing in whole blood from spinal muscular atrophy patients and healthy control infants Overall design: Here, we performed whole-blood RNA sequencing and differential expression analyses in SM

NIAID Data Ecosystem60

SMCHD1 activates the expression of genes required for the expansion of human myoblasts (RNA-seq). SMCHD1 activates the expression of genes required for the expansion of human myoblasts (RNA-seq)

肌肉疾病分子机制

转录组功能分析

In human muscle, SMCHD1 mutations are associated with the onset of FSHD2, but the mechanism driving the disease onset remains unclear. A commonly accepted explanation is the loss of SMCHD1 binding to

NIAID Data Ecosystem40

E-MEXP-3761

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