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Transcriptome analysis of cardiac hypertrophy
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创建时间：
2013-11-30
相关数据集
Additional file 1: of Muscle-specific regulation of right ventricular transcriptional responses to chronic hypoxia-induced hypertrophy by the muscle ring finger-1 (MuRF1) ubiquitin ligase in mice
Table S1. Differentially expressed genes in MuRF1â/â mice challenged with hypoxia (XLSX 59 kb)
NIAID Data Ecosystem90
Differential expression between heart failure and healthy CVS tissues.
The table shows the results from differential expression analysis of genes and isoforms between pre-LVAD samples and the three healthy CVS tissues (iPSC-CVPC, adult heart and adult arteria). Columns A
NIAID Data Ecosystem50
RNA Expression Profiling of Human iPSC-Derived Cardiomyocytes in a Cardiac Hypertrophy Model [miRNA expression]
RNA Expression Profiling of Human iPSC-Derived Cardiomyocytes in a Cardiac Hypertrophy Model [miRNA expression]
ChEBI2014-10-03 更新30
Augmented sphingosine 1 phosphate receptor-1 signaling in cardiac fibroblasts induces cardiac hypertrophy and fibrosis through angiotensin II and interleukin-6
Background: Cardiac fibroblasts, together with cardiomyocytes, occupy the majority of cells in the myocardium and are involved in myocardial remodeling. The lysophospholipid mediator sphigosine-1-phos
NIAID Data Ecosystem60
Genome-Wide Gene Expression Analysis Shows AKAP13-Mediated PKD1 Signaling Regulates the Transcriptional Response to Cardiac Hypertrophy
In the heart, scaffolding proteins such as A-Kinase Anchoring Proteins (AKAPs) play a crucial role in normal cellular function by serving as a signaling hub for multiple protein kinases including prot
NIAID Data Ecosystem50
